SubMorphic – Comprehensive Premature Ejaculation Treatment (Morphic Field)

This energetic field is based on scientific research and works to resolve the issue via multiple approaches. The document below gives a breakdown:

https://docs.google.com/document/d/1NQEZqpjngZYJiUX_wogF_A_JcxqIycmF/edit

5 to 10 min per day should suffice

Comprehensive Premature Ejaculation Treatment:

Selective Serotonin Reuptake Inhibitors (SSRIs):

Concept:

Serotonin or 5-hydroxytryptamine (5-HT) is a monoamine neurotransmitter (a messenger chemical that carries signals between nerve cells in the brain). It’s thought to have a good influence on mood, emotion and sleep.

In humans, serotonin is the neurotransmitter most clearly associated with ejaculation.

5-HT receptor subtypes have a role in ejaculation: 5-HT1A and 5-HT2C receptors.

Activation of postsynaptic 5-HT2C receptor prolongs ejaculatory latency, whereas activation of presynaptic 5-HT1A auto-receptor, which inhibits 5-HT release, decreases ejaculatory latency.

Selective Serotonin Reuptake Inhibitors (SSRIs) work by preventing cells from reabsorbing (reuptaking) serotonin. This means that the signals caused by serotonin are extended, so serotonin is available to pass further messages between nearby nerve cells.

All SSRIs, inhibit reuptake of serotonin into presynaptic serotonergic neurons, an action that increases the availability of serotonin at the synapse and ultimately, enhances serotonergic function in the central nervous system (CNS).

This mechanism of action depends on the binding of the drug to serotonin transporter protein. It has also been suggested that the efficacy of SSRIs in inhibiting PE, is probably due to increase synaptic 5-HT concentrations via blockade of the 5-HT transporter and activation of the 5-HT2 C receptor, which then decreases the function of the 5-HT1A receptor.

SSRIs like: Paroxetine and Sertraline have provided significant benefits in patients with PE, either via daily administration or on-demand use before intercourse.

They are also used to treat Depression, Obsessive Compulsive Disorder and Panic Attacks.

In study, 480 patients with mean age of 36.5 years were studied. In Paroxetine (an SSRI), timing increased from 71.5±69.1 to 379.9±154.3 second in span of 8 weeks.

Another study from 1998, trusted source examined the effects of Sertraline (an SSRI) on 46 men who experienced PE. They ranged in age from 22 to 63.

Researchers found that a 100-mg dose resulted in an average ejaculatory interval of 16.4 minutes.

Working:

Extremely low expression of 5HT1-A auto-receptor.

Overexpression of 5-HT2C receptor.

Mimic 100 mg energetic dose of Sertraline HCl (an SSRI) with all the benefits minus all the side effects.

Mimic 20 mg energetic dose of Paroxetine HCl (an SSRI) with all the benefits minus all the side effects.

Topical therapy (anaesthetics):

Concept:

Lidocaine-prilocaine 5% cream:

Topical anaesthetics have been used for treatment of PE to decrease penile stimulation, and thus delay the time to ejaculation. The application of lidocaine-prilocaine 5% cream to penis before covering it with a condom 20–30 min before intercourse delayed ejaculation in men with PE.

Lidocaine-prilocaine cream used for a period of 30–60 days significantly increases the mean IELT (Intra-Vaginal Ejaculation Latency Time), especially when penile hypersensitivity is likely to be the cause.

Working:

Mimic energetic administration of Lidocaine-prilocaine 5% cream on the glans of the penis.

Phosphodiesterase type 5 inhibitor:

Concept:

Several clinical trials have examined the potential effectiveness of the PDE5 inhibitor, Sildenafil, in the treatment of PE. The rationale for the use of PDE5 inhibitors in the treatment of PE may be due to peripheral and/or central mechanisms. Ejaculation retarding by peripheral actions may include modulation of contractile response of vas deferens, seminal vesicles, prostate and urethra, induction of a state of peripheral analgesia, and prolongation of the total duration of erection. Central mechanisms may involve lessening of the central sympathetic output.

The on-demand administration of 50–100 mg Sildenafil citrate for 1–3 months, in patients with PE complicated by ED, was found to safely and effectively improve erectile function and prolong ejaculation.

Working:

Mimic 100 mg Sildenafil Citrate energetically.

Opioid agonist:

Concept:

Tramadol is a centrally acting analgesic with two mechanisms of action. It exerts an effect on the μ-opioid receptor which reduce sensitivity, but also inhibits noradrenaline and serotonin reuptake.

Administration of 50 mg tramadol HCl 2 h before planned sexual activity for 8 weeks, resulted in a significant increase of IELT and intercourse satisfaction.

Working:

Mimic 50 mg Tramadol HCl energetically.

Adrenergic Nerve Blockade:

Concept:

Adrenergic nerve blockade has been proposed as a treatment for PE.

Silodosin, a highly selective α1A-adrenoceptor antagonist and on-demand use of 4 mg silodosin orally 1 h before sexual intercourse in treatment of patients with PE was effective in improving PE profile and the IELT. The treatment was based on the fact that emission and ejaculation are under the influence of the sympathetic nervous system.

Working:

Mimic 4mg dose of Silodosin energetically.

Folic Acid:

Concept:

Folic acid provides the methyl group for the conversion of methionine to S-adenosylmethionine, which itself has been shown to influence serotonin metabolism. It has an important role in the synthesis of tetrahydrobiopterin, the rate limiting step in the synthesis of dopamine, noradrenaline and serotonin.

Folic acid supplementation was reported to produce an antidepressant-like effect, mediated by an interaction with the noradrenergic receptors (α1 and α2) and serotonergic receptors (5-HT1A and 5-HT2A/2 C).

Working:

Mimic 1000 mcg dose of Folic Acid (Vitamin B9) energetically.

Surgical Treatments:

Concept:

a. Glans augmentation:

Glans augmentation has been a technique proposed to desensitise the glans penis and slow the ejaculatory reflex. It is a method in which hyaluronic acid is injected into the glans at the coronal edge to provide analgesia of the penis. Hyaluronic acid is a glycosaminoglycan and bulking agent that has been used to insulate the nerve endings and provide long-term (>1 year) local anaesthesia. It was reported to increase the IELT and satisfaction in patients with PE.

b. Pulsed radiofrequency neuromodulation

Pulsed radiofrequency neuromodulation has been used for treatment of PE by desensitisation of the dorsal penile nerves. It showed a significant increase in the IELT in patients with PE. There were no reported side-effects post-procedure such as pain, penile hypoesthesia, or ED.

Working:

Mimic Glans Augmentation surgery safely.

Mimic Pulsed Radiofrequency Neuromodulation surgery safely.

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